(VIII) 26-year-old patient with pulmonary sarcoidosis, pericarditis and suspition of cardiac sarcoidosis Justyna Błaut-Jurkowska MD, Lidia Tomkiewicz-Pająk MD, PhD, Izabela Karch MD, Joanna Łuszczak MD, Mariusz Urban MD, Aleksandra Lenart MD, Prof. Jacek Musiał, Prof. Maria Olszowska, Prof. Piotr Podolec
EXPERTISE 1 EXPERTISE 2 “Development of the European Network in Orphan Cardiovascular Diseases” “Rozszerzenie Europejskiej Sieci Współpracy ds. Sierocych Chorób Kardiologicznych”
(VIII) 25 year old man with hyperkinetic circulation Jakub Stępniewski, Grzegorz Kopeć, Prof. Piotr Podolec
EXPERTISE 1 EXPERTISE 2 EXPERTISE 3 “Development of the European Network in Orphan Cardiovascular Diseases” “Rozszerzenie Europejskiej Sieci Współpracy ds. Sierocych Chorób Kardiologicznych”
(VIII-3) 24-year-old patient with the eosinophilic myocarditis in the course of the ascariasis. D. Kudliński MD, G. Kopeć MD, PhD, Sylwia Wiśniowska-Śmiałek MD, M. Urbańczyk MD, M. Krupiński MD, P. Rubiś MD, PhD, A. Leśniak-Sobelga MD, PhD, Prof. M. Kostkiewicz MD, PhD, Prof. P. Podolec MD, PhD
Background Ascariasis is the most frequent parasitic disease in the world. It is caused by Ascaris lumbricoides, the human roundworm which can reach a length of up to 45 cm with a diameter of 5 mm. In the course of ascariasis the reactive eosinophilia is usually present. Cardiac disease is the major cause of death in the course of sustained eosinophilia, whether reactive or clonal. Eosinophilic myocarditis is a rare, potentially fatal disease. Cardiac involvement does not correlate with the …
(VII-VIII-4) Acute thromboembolic disease complicated with heparin induced thrombocytopenia type II in pregnant woman Leszek Drabik. MD, Agata Leśniak-Sobelga MD, PhD, Lidia Tomkiewicz-Pająk MD, PhD, Magdalena Kostkiewicz MD, PhD, Maria Olszowska MD, PhD, Prof. Piotr Podolec MD, PhD, Sylwia Wiśniowska –Śmiałek MD.
Background Heparin induced thrombocytopenia type II (HIT-II) is immune-mediated disorder with life-threatening prothrombotic complications. HIT-II is caused by formation of antibodies which activate the platelets following heparin administration [1]. The incidence of HIT-II is lower in pregnant than in nonpregnant women [2]. Several features of the history and physical examination support a diagnosis of HIT-II [3]: -fall in platelet count ≥50%, from highest platelet count after heparin exposure, in 10% of cases platelet count fall is 30-50% -fall in platelet …